Vesicular Monoamine Transporters

Arrowheads inDindicate white areas of triple colocalized FcRn, IgG, and LAMP1, a phenomenon seen in 50% of cells investigated, and the arrow indicates yellow area of FcRn colocalized with LAMP1

Arrowheads inDindicate white areas of triple colocalized FcRn, IgG, and LAMP1, a phenomenon seen in 50% of cells investigated, and the arrow indicates yellow area of FcRn colocalized with LAMP1.…

Continue ReadingArrowheads inDindicate white areas of triple colocalized FcRn, IgG, and LAMP1, a phenomenon seen in 50% of cells investigated, and the arrow indicates yellow area of FcRn colocalized with LAMP1

In seronegative pediatric individuals, there’s a high frequency from the antibody against myelin oligodendrocyte glycoprotein (MOG-IgG) indicating another disease group, however the clinical distinction between both of these diseases may be challenging

In seronegative pediatric individuals, there's a high frequency from the antibody against myelin oligodendrocyte glycoprotein (MOG-IgG) indicating another disease group, however the clinical distinction between both of these diseases may…

Continue ReadingIn seronegative pediatric individuals, there’s a high frequency from the antibody against myelin oligodendrocyte glycoprotein (MOG-IgG) indicating another disease group, however the clinical distinction between both of these diseases may be challenging

Components from mouse liver and human being adipose cells were used while positive settings for PPAR- and -, respectively

Components from mouse liver and human being adipose cells were used while positive settings for PPAR- and -, respectively. PPAR- and - (co)agonists might be of restorative interest for the…

Continue ReadingComponents from mouse liver and human being adipose cells were used while positive settings for PPAR- and -, respectively

We hypothesized that ER upregulation by Sirt6 was due to protein stabilization via proteasome inhibition and repeated the experiments in the presence of the proteasome inhibitor MG132

We hypothesized that ER upregulation by Sirt6 was due to protein stabilization via proteasome inhibition and repeated the experiments in the presence of the proteasome inhibitor MG132. ER stabilization promoted…

Continue ReadingWe hypothesized that ER upregulation by Sirt6 was due to protein stabilization via proteasome inhibition and repeated the experiments in the presence of the proteasome inhibitor MG132

It was discovered that in the absence of miR-124 in SK-N-SH neuroblastoma cells, AhR protein expression increases while cell proliferation slows and the cell cycle is blocked [65]

It was discovered that in the absence of miR-124 in SK-N-SH neuroblastoma cells, AhR protein expression increases while cell proliferation slows and the cell cycle is blocked [65]. 5. therapeutic…

Continue ReadingIt was discovered that in the absence of miR-124 in SK-N-SH neuroblastoma cells, AhR protein expression increases while cell proliferation slows and the cell cycle is blocked [65]